TY - JOUR
T1 - Encephalopathy mimicking non-convulsive status epilepticus
AU - Nandhagopal, Ramachandiran
AU - Al-Murshedi, Fathiya
AU - Al-Busaidi, Mujahid
AU - Al-Busaidi, Amna
N1 - Publisher Copyright:
© 2018, Saudi Arabian Armed Forces Hospital. All rights reserved.
PY - 2018/1
Y1 - 2018/1
N2 - Primary hyperammonemic encephalopathy due to urea cycle disorders (UCD) typically manifests with episodic unresponsiveness and this clinical entity is not often included in the differential diagnosis of presumed non-convulsive status epilepticus (NCSE) However, this diagnostic consideration has therapeutic implications. In this report, we document the therapeutic importance of elucidating the specific cause of hyperammonemic encephalopathy that closely mimicked NCSE through 2 unique illustrative cases.
AB - Primary hyperammonemic encephalopathy due to urea cycle disorders (UCD) typically manifests with episodic unresponsiveness and this clinical entity is not often included in the differential diagnosis of presumed non-convulsive status epilepticus (NCSE) However, this diagnostic consideration has therapeutic implications. In this report, we document the therapeutic importance of elucidating the specific cause of hyperammonemic encephalopathy that closely mimicked NCSE through 2 unique illustrative cases.
UR - http://www.scopus.com/inward/record.url?scp=85041026833&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=85041026833&partnerID=8YFLogxK
U2 - 10.17712/nsj.2018.1.20170214
DO - 10.17712/nsj.2018.1.20170214
M3 - Article
C2 - 29455222
AN - SCOPUS:85041026833
SN - 1319-6138
VL - 23
SP - 52
EP - 56
JO - Neurosciences
JF - Neurosciences
IS - 1
ER -
