Encephalopathy mimicking non-convulsive status epilepticus

Research output: Contribution to journalArticle

Abstract

Primary hyperammonemic encephalopathy due to urea cycle disorders (UCD) typically manifests with episodic unresponsiveness and this clinical entity is not often included in the differential diagnosis of presumed non-convulsive status epilepticus (NCSE) However, this diagnostic consideration has therapeutic implications. In this report, we document the therapeutic importance of elucidating the specific cause of hyperammonemic encephalopathy that closely mimicked NCSE through 2 unique illustrative cases.

Original languageEnglish
Pages (from-to)52-56
Number of pages5
JournalNeurosciences
Volume23
Issue number1
DOIs
Publication statusPublished - Jan 1 2018

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Status Epilepticus
Brain Diseases
Inborn Urea Cycle Disorder
Differential Diagnosis
Therapeutics

ASJC Scopus subject areas

  • Clinical Neurology
  • Psychiatry and Mental health

Cite this

Encephalopathy mimicking non-convulsive status epilepticus. / Nandhagopal, Ramachandiran; Al-Murshedi, Fathiya; Al-Busaidi, Mujahid; Al-Busaidi, Amna.

In: Neurosciences, Vol. 23, No. 1, 01.01.2018, p. 52-56.

Research output: Contribution to journalArticle

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